Mildred L. Brogdon, the beneficiary in three insurance policies providing for indemnity for death by accidental means, issued by The National Life and Accident Insurance Company on the life of her husband, James Phillip Brogdon, recovered judgment from which the Insurance Company appeals.
Two of the policies provided for ordinary death benefits, and the amounts therefor specified were paid. The indemnity provided for in the three policies is payable if the insured sustained bodily injuries effected solely through external, violent, and accidental means, and that such bodily injuries have directly and independently of all other causes, caused the death of the insured within ninety days from the time such injuries were so sustained, with the provision that the benefit shall not be payable “if death results directly or indirectly” from bodily or mental infirmity or disease. Brogdon died on August 20, 1957.
The jury found: The deceased sustained bodily injuries on August 20, 1957; these injuries were effected solely through violent, external and accidental means; these injuries caused the death of Brogdon directly and independently of all other causes; on or about August 20, 1957, Brogdon was suffering from a defect in his metabolism ; such defect was a contributing cause of his death; and the defect in his metabolism was effected solely through violent, external, and accidental means.
Appellant contends that there was no evidence, or the evidence was insufficient, to support the findings that any injuries the deceased may have sustained were effected solely through external, violent, and accidental means; or that his death resulted directly and independently of all other causes, from bodily injuries effected solely through external, violent, and accidental means; or that the metabolic defect from which deceased was suffering was effected solely through external, violent, and accidental means; further, that there is an irreconcilable conflict between the finding that bodily injuries caused death, directly and independently of all other causes, and the finding that a metabolic defect was a contributing cause.
At the time of his death, Brogdon was working as a mechanic for.a trucking company. He was 24 years of age, 6 feet 3 inches in height, and weighed about 170 pounds. He was healthy and strong. At approximately 10 :00 A. M. on August 20, *405 1957, he was “airing up” a truck tire; “He was bent over a tire. * * * Yes, kinda squatted down there by the tire.” A coem-ployee, Leonard Johnson, approached him from the rear and jabbed, or “goosed”, him in the ribs; Johnson ran; he testified that he knew Brogdon would try to catch him; Brogdon jumped up and ran toward Johnson, overtaking him after two of three steps; Brogdon caught Johnson from the back and got hold of him; they scuffled momentarily; Brogdon’s grip on Johnson relaxed and he fell to the ground and died. A physician made an incision in Brogdon’s chest and performed a cardiac message, but he was not revived.
About three hours after Brogdon’s death Dr. Irvine, a pathologist, performed an autopsy. He found nothing that indicated a cause of death. According to his testimony, Brogdon was extremely well developed and a very muscular man; his heart was that of an extremely healthy man; his lungs were normal; so were his liver, gall bladder, spleen, pancreas, gastrointestinal tract, kidneys, bladder, and adrenal glands. Dr. Irvine found a slightly increased hyperplastic lymphoid tissue in the thymus gland, which he said was “a slight degeneration, but not too much, just a very little, which would indicate that some metabolism had taken place. It is possible that some metabolism had taken place shortly prior to his death.” From a complete anatomical investigation, Dr. Irvine found no visible alteration of the body that would indicate a cause of death. He said there were causes of death which cannot be detected by an autopsy.
He further testified in substance: He thought death was caused by a coronary artery spasm or by ventricular fibrillation, the probability being that it was the former ; a coronary spasm is “a status of complete contraction of the arterial wall muscles causing closure of the opening in the center of the vessel and occluding the flow of blood”; ventricular fibrillation is an actual physiological state of the heart muscle in which a disorganized rhythm becomes established, which is in itself incompatible with life, and is due to an abnormality in the metabolism or conduction of the heart muscle. In such cases the heart muscle does not get the proper chemicals, the lack of which causes it to have a disorganized rhythm. Generally, the things which bring about these coronary disturbances are overeating, overexertion, or excitement. There are many possible causes of metabolic imbalance; physical labor and diet, such as not enough or too much salt, may enter into it; it is a very common thing; it is not unusual or even particularly abnormal to have a temporary imbalance; it occurs in many normal human beings; it is the usual process that a person gets in balance again after he becomes temporarily imbalanced following stress, strain and excitement or overeating; the imbalance can be created by hard work, regular manual labor and strenuous exertion. Hard labor and strenuous exertion of deceased were contributing factors to his condition. “If he had not sustained the sudden excitement or sudden stress, which was described, he would not have died with a metabolic defect as it was.” He believed it was a spasm of the artery going into his heart that caused his death. “Q. * * * in all reasonable probability, based upon the facts which you have given us, would it have been some traumatic occurrence that brought about the death of Phillip Brog-don? A. With some added insult, yes, sir. Q. Without that added insult would he have died? A. Not under the existing circumstances, no, sir.”
We think the evidence is sufficient to support the findings that deceased sustained bodily injuries which were effected solely through external, violent, and accidental means, and that those injuries caused his death, directly and independently of all other causes. Bryant v. Continental Casualty Co.,
We quote from Hanna v. Rio Grande Nat. Life Ins. Co., Tex.Civ.App.,
The policy provisions considered here have been applied to various states of fact, and the decisions are not harmonious. Some cases hold that if the accident accelerates death, it must be held to be the proximate and sole cause, even though death was hastened by a diseased condition. Fidelity & Casualty Co. v. Meyer,
Some cases make a distinction between proximate and remote cause, holding that the remote cause will not prevent recovery, and that in cases of doubt as to whether an existing infirmity or disease is a remote or proximate cause, the issue is one for the jury. In Equitable Life Assur. Soc. of United States v. Gratiot,
The court, in Bird v. St. Paul Fire & Marine Ins. Co.,
" * * * if, under the peculiar temperament or condition of health of an individual upon whom it is inflicted, such injury appears as the active, efficient cause that sets in motion agencies that result in death, without the intervention of any other independent force, then it should be regarded as the sole and proximate cause of death. The fact that the physical infirmity of the victim may be a necessary condition to the result does not deprive the injury of its distinction as the sole producing cause. In such case, disease and low vitality do not rise to the dignity of concurring causes, but, in having deprived nature of her normal power of resistance to attack, appear rather as the passive allies of the agencies set in motion by the injury.” Driskell v. United States Health & Accident Ins. Co.,
Policy provisions substantially identical to those here involved were considered in Equitable Life Assur. Soc. of United States v. Gratiot,
“Counsel’s claim that no recovery should be permitted herein would, doubtless, be valid, if it were conceded that any bodily infirmity which, from a scientific standpoint, contributes to the death, would have that effect under the terms of the contract, and that is virtually what counsel, upon ultimate analysis, insist on. They apparently overlook the difference between proximate cause on the one hand, and remote cause or a condition on the other, as recognized in law, not bearing in mind, seemingly, that a contributing cause from a scientific standpoint is not necessarily a proximately contributing cause from a legal standpoint. Silverstein v. Metropolitan Life Insurance Co., supra [254 N.Y. 81 ,171 N.E. 914 ]. The foregoing cases as a whole, or at least many of them, all dealing with policies similar to that in the case at bar, clearly show, we think, that, in solving the problem before us, we cannot overlook the distinction between proximate and remote cause, and that if a disease, *408 bodily infirmity, or predisposing cause in fact existed, as claimed, still unless it can be said to be one of the proximate causes instead of only the remote cause or condition, recovery should not, on that account, be denied. We said in Lemos v. Madden,28 Wyo. 1 , 10, 12, 16,200 P. 791 , that proximate cause is probable cause, and remote cause is improbable cause; that the mere fact that one cause may in point of time concur with another does not necessarily show that it is an efficient cause, and may still be considered a remote cause, and that a cause may, in law, be a remote cause, notwithstanding the fact that, but for its existence, no injury would have occurred.”
In Runyon v. Commonwealth Casualty Co., 9 N.J.Misc. 487,
What we have said is applicable if we assume that deceased was afflicted with bodily infirmity or disease, not caused by the accident, which contributed to his death. That he was not so afflicted might well be found from the evidence. His condition was described as a “state” or “status”; it is a very common thing; it is not unusual or particularly abnormal; it occurs in many normal human beings.
“It is not every ailment, indisposition, or imperfection that renders one of unsound health. There must be a material departure from sound health * * *.” McVeigh v. International Travelers Assur. Co., Tex.Civ.App.,
Moreover, the jury found that deceased’s, metabolic defect was caused solely by violent, external, and accidental means, which,, we think, shows there was no conflict between the findings. The evidence supporting that finding has substance and weight.. According to Dr. Irvine, such metabolic-defect is generally caused by overeating, overexertion, or excitement. There was. no evidence of overeating. Overexertion and excitement were shown by evidence of' probative force. It is possible that improper diet might contribute to such a. condition; but that was not shown here. His wife said he did not like salt, but she-did not limit salt in his food, and there-is no evidence that he consumed less salt than the average person. We think the evidence is sufficient to support the finding-that the metabolic defect was caused by the-accident. If so, its contribution to the fatal result would not preclude recovery. Robinson v. Aetna Life Ins. Co., Tex.Com.App.,
The judgment is affirmed.
